1- Discuss the role of pulmonary surfactant in the pathogenesis of atelectasis and explain the interpulmonary shunting with the associated ventilation perfusion mismatching that occurs with the resulting hypoxemia.
2-Discuss hypoxemic Respiratory Distress Syndrome and the management of the body systems for a patient in acute failure.
3- Relate specific neuromuscular diseases of the musculature, peripheral motor nerves, neuromuscular junction, and spinal cord and indicate the RCP’s role in assessing progression of these diseases.
Differentiate idiopathic pulmonary fibrosis from hypersensitivity pneumonitis in this typically restrictive pulmonary disease. Include in your discussion how a diagnosis is made, its management, course, and prognosis might be related to a patient suffering from this pervasive illness.
5- Discuss the pathogenesis of pneumocystis carinii (Jervecki) in an immuno-compromised host and relate a treatment and prophylactic approach in minimizing the effects of this debilitating diseasses.
6-Differentiate small-cell carcinoma from the three non-small-cell lung cancers and indicate the most important risk factor for developing bronchogenic carcinoma.
7-Indicate the relation of secondary tuberculosis to a concomitant infection with human immunodeficiency virus and relate a therapeutic approach in treating patients with the virus.
8-Distinguish between cor pulmonale and pulmonary hypertension and explain a relation to pulmonary fat emboli.
9-Indicate the pathophysiological similarity between infant respiratory distress and adult respiratory distress. Determine several conditions that make them different, and indicate three methods of treatment that could potentially make them better.
10-Describe the pathology with accompanying signs, symptoms, and prognosis for three fungal lung diseases. Include in your discussion several clinical manifestations and treatment regimes in improving pulmonary complications.
11- Describe the relation of the KVEIM antigen and the accompanying etiology, pathogenesis, and treatment of sarcoidosis.
12-Explain non-cardiogenic pulmonary edema in terms of fluid absorption at the pulmonary capillary membrane.
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